Glucose-6-Phosphate
Dehydrogenase
3D
Image of G6PD:
Image borrowed from http://en.wikipedia.org/wiki/Image:G6PD.png
Brief Background
Glucose-6-Phosphate Dehydrogenase
(G6PD) is an enzyme involved in the first step of the pentose phosphate
pathway, producing the 5-carbon sugar, ribose, for later nucleic acid synthesis
and fatty acid synthesis. G6PD catalyzes the oxidation/reduction reaction from
glucose-6-phosphate to 6-phophogluconate. This reaction pulls two electrons
from G6P in order to reduce the coenzyme NADP+ to NADPH.
Reaction Mechanism of
G6PD for the Pentose Phosphate pathway:
Image borrowed from http://en.wikipedia.org/wiki/Image:G6PD_mechanism.png
On a Molecular Level
The
G6PD gene can be found on the chromosome Xq28 region. With 13 exons and 12 introns, the actual
gene coding for G6PD is about 18,500 base pairs long. In its active form, it is
made up of either two (dimer) or four (tetramer)
identical subunits, each with a molecular mass of about 59-kilodaltons.
Determined from cDNA sequencing, the primary
structure contains 515 amino acids.
Comparison between G6PD from yeast and leuconostoc mesenteroides:
Several
studies have been done comparing G6PD in humans, yeast, and leuconostoc mesenteroides. Below is a chart
comparing data from yeast and L.mesenteroids. The molecular weight of G6PD in L.mesenteroids is
about 104 Kd, and consists
of two subunits of about 55 Kd. The G6PD in this
species is unique in that it has no cysteine/cysteine
residues and has an essential lysine at the active site. It also exhibits a dual coenzyme specificity, for both NAD+ and NADP+, while
Yeast and most other types of G6PD only exhibit one, just NADP+.
|
Yeast |
L.mesenteroids |
Co-factors |
NADP+ |
NAD+, NADP+ |
Optimum pH |
9.2 |
7.0-8.5 |
Optimum
Temperature |
~55oC |
~50oC |
pI |
6.1 |
4.6 |
Stability |
Stable pH 5-11 |
Stable pH 6-9 |
Effectors |
Mg2+ |
HCO-3 |
Inhibitors |
Neucleoside monophosphates
Some nucleoside di- and triphosphates
Myristic acid Dihydroepiandrosterone Palmitoyl-CoA |
Phosphate ATP is a
competitive Inhibiton NADPH is competitive
in the NAD+- dependent Reaction |
Real World Application:
G6PD Deficiency Disorder
Deficiency
in G6PD is a hereditary X-linked recessive disorder that mostly found in men,
and frequently occurs in African Americans, Asians, and Mediterranean peoples. G6PD
is found in red blood cells, and when G6PD in these erythrocytes cannot
maintain NADPH levels, which in turn maintains glutathione levels, the red
blood cells lose protection against oxidative damage. When oxidative stress
occurs, red blood cells often rupture, leading to hemolytic anemia. Other
clinical symptoms include neonatal jaundice leading to kernicterus,
favism - or haemolysis
after the ingestion of fava beans, and, in severe
cases, chronic non-sphericytical haemoolytic
anaemia.
This
recessive disorder has stayed within the genotypes of humans most probably
because the deficiency has been found to protect against a very lethal form of
malaria.
To be continued,
updated, and revised… ^__^
Sources:
1.
http://rialto.com/g6pd/ G6PD deficiency
homepage
2. www.cchi.com.hk/
specialtopic/case1/case1.htm
3.
http://www.worthington-biochem.com/ZF/default.html
4.
http://www.serva.de/products/knowledge/061334.shtml
5.
http://www.answers.com/topic/glucose-6-phosphate-dehydrogenase-deficiency#copyright